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It is possible that bypassing the inhibitory actions of IGF-binding proteins with IGF-I LR3 may allow for better-supported activation of IGF signaling pathways that contribute to a reduction in contraction-induced damage

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Accordingly, we suggest that communication should focus on uncertainty managementneither exaggerating teratogenic risks nor downplaying the metabolic and pregnancy costs of post-discontinuation rebound

Similarly, Trx2 has been shown to negatively regulate apoptosis across multiple studies (173,174)