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These conditions are not contraindications to semaglutide or tirzepatide use

With triple receptor agonism maintaining appetite suppression and increased energy expenditure, a new equilibrium takes longer to reach than with less potent agents

In the pancreas, semaglutide stimulates insulin secretion in a glucose-dependent manner meaning it triggers insulin release when blood glucose is elevated, but not during normoglycemia, which substantially reduces the risk of hypoglycemia compared to older antidiabetic agents

Think of NF-kB sort of like a firefighter, it sits inside cells, waiting for a signal that something is wrong (like an injury, an infection, or stress) only then does it activate and tell the cell how to defend itself