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Specific mechanisms regarding how these mutations regulate the activity of GDH are unclear
In a range of AD animal models, treatment with GLP-1, GIP, or dual-GLP-1/GIP receptor agonists effectively protected cognition, synaptic trans-mission, long-term potentiation (LTP), and prevented the loss of synapses and neurons
Identity confirmation has failed
This treatment is commonly used to address deficiencies caused by poor diet, malabsorption issues, or certain medical conditions such as pernicious anemia
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