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Furthermore, our data suggest potent inhibition of the FAT helix 2-3 site is sufficient for cellular disruption of the FAK-paxillin interaction, which is in alignment with previous reports characterizing the interaction 25

Development of a fragment-based screening assay for the focal adhesion targeting domain using SPR and NMR

Also, why leave the patient perhaps 2 weeks without their treatment when they need it

Because the bulk of published research investigating these two compounds in combination has used roughly equal proportions, and because there's no strong mechanistic reason to weight angiogenesis (BPC-157) over cell migration (TB-500) in tissue-repair research