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The knockdown of Hbs1 expression by dsHbs1 treatment results in the reduced Pelo protein level but not its mRNA level
Damage to endothelial cells results in heightened secretion of the vasoconstrictor endothelin and diminished secretion of the vasodilator nitric oxide, causing sustained renal vasoconstriction, impaired renal perfusion, and aggravated renal injury [83]
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