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The observations from our animal studies prompted us to examine whether dysregulated cellular glutathione homeostasis could render hepatocytes susceptible to TNF-induced cell death
A glutathione-Nrf2-thioredoxin cross-talk ensures keratinocyte survival and efficient wound repair
These metabolites are critical regulators of redox homeostasis, energy balance, and amino acid metabolismkey processes that are frequently disrupted in chronic metabolic diseases
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