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Lamark, T
This is in sharp contrast to GSH, which can enter cells only slowly via specific transporters
The metabolic drug product or metabolite produced following phase I modifications can also act as a substrate for additional modifications, such as phase II conjugation reactions
Although, the molecular mechanisms responsible for the anti-inflammatory effects of GLP-1/GLP-1R signals are still under investigation, the PKA/CREB pathway axis and associated active components may be major signals responsible for the anti-inflammatory effects of GLP-1/GLP-1R [23]
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