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Furthermore, the precise mechanisms by which GLP-1RAs confer their cardioprotective effects remain incompletely understood, necessitating further investigation

These substances not only induce physical dependence and psychological addiction but also contribute to severe neurotoxicity, manifesting as persistent cognitive impairment, emotional instability, and motor dysfunction

But that peripheral-only action means it does not suppress appetite, does not change energy expenditure through central pathways, and does not produce the dramatic caloric deficits that GLP-1 agonists like semaglutide or tirzepatide create

Deficits of spatial learning and working memory in spontaneously hypertensive rats