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This sensitization to TGF-beta signaling augments expression of matrix genes including collagens, fibronectin, and proteoglycans, while appropriate regulation prevents excessive TGF-beta activity associated with pathological fibrosis

At the protein level, FOXO4-DRI apparently reduced markers tied to myofibroblasts and matrix deposition, including -SMA and Col1a1

GHK-Cu demonstrates wound healing, anti-aging, and gene modulation effects in isolation n.d

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