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In nucleated cells, apoptosis and necroptosis dont occur simultaneously, with each inhibiting the other one [26]
In addition, FAK has been proposed as a target for fibrosis, due to its upregulation in fibrotic diseases 6,7 and role in modulating myofibroblast transformation, extracellular matrix (ECM) remodeling, and fibroblast migration 8,9
However, its essential to consult a dermatologist before starting any treatment
They play a role in the pathophysiology of the last of these by binding to their receptors in the brain to stimulate the release of cytokines, which lead directly to the formation of insoluble amyloid senile plaques and neuronal damage
