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Deficiency in the level of activity of glucose-6-phosphate dehydrogenase (G6PD) is the basis of favism, primaquine (an anti-malarial drug) sensitivity and several other drug-sensitive hemolytic anemias, anemia and jaundice in the newborn, and chronic nonspherocytic hemolytic anemia

Since a batteries of free radicals scavenging enzyme system depend directly on availability of GSH for detoxification of ROS, molecules capable of modulating glutathione biosynthesis could potentially protect free radicals mediated neurodegenration under hypoxic condition

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HIV alters DC intracellular mechanisms to prevent NK cell-mediated apoptosis by upregulating cellular FLICE-like inhibitory protein (c-FLIP) and IL-10, thereby mediating protection