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IGF-1 availability is tightly regulated by the so-called insulin- like growth factor binding proteins (IGFBPs), which may act by increasing IGF-1 half-life, from minutes to hours (most commonly by forming a tertiary complex with Acid-Labile Subunit and IGFBP3), however blocking its binding to the insulin-like growth factor 1 receptor (IGF- 1R) [7]
Czerninski R, Zini A, Sgan-Cohen HD
In this review, we discuss the phenomenon of ADE, its possible mechanism that may play a significant role in the pathogenesis of COVID-19 by relating the previous and some current findings with the COVID-19 pandemic and highlights the interplay of antibodies that may be neutralizing or non-neutralizing in nature with viral surface receptors that may lead to this condition
