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The result is impaired BH4 recycling less BH4 available to maintain eNOS in its coupled, NO-producing configuration independent of the direct homocysteine-mediated BH4 oxidation described above
Oxidative stress caused by the overproduction of ROS and reactive nitrogen species, as well as changes in antioxidant enzyme activities, is implicated in NEO-induced injury to cellular molecules such as lipids, DNA, and proteins (Wang et al
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doi: 10.1016/S0140-6736(21)00536-5 15 GutniakMrkovCHolstJJAhrnBEfendiS
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