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Mitochondrial ROS production and oxidative mtDNA damage is increased during reoxygenation in the GPX1 knockout mice, in addition to structural abnormalities in cardiac mitochondria and myocytes, suggesting GPX1 may play an important role in protecting cardiac mitochondria from reoxygenation damage in vivo
Hevesi Z et al
Biological evaluations included both in vitro and in vivo tests
DIANN main report file was processed into R using the diann package and the MaxLFQ normalization algorithm while applying a 1% q-value filter on both precursors and protein groups
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