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In a mouse model of stroke, intracerebroventricular sodium selenate treatment elevates GPX4 levels by activating transcription factors activating enhancer binding protein 2 and specificity protein 1, while also providing protection against excitotoxicity and endoplasmic reticulum stress-induced cell death independent of GPX4 (102)
Given the considerable continued growth expected in the GLP-1 pipeline, plus a new indication and a generic expected as early as December 2024, Prime Therapeutics talented team of clinical experts has developed this publication to focus on the GLP-1 emerging landscape
22 press release
This hints at the possibility of these monomers acting as innovative ferroptosis inducers by regulating GSH content
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