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For GHK-Cu, the best-supported claims come from topical and wound-related evidence, while injectable and systemic claims need more rigorous human data [2], [3], [8]
Liver biopsies obtained from both NASH patients and rodent NAFLD models demonstrated defective mitochondrial respiratory chain function together with altered mitochondrial structure, presented as enlarged mitochondria, rounded cristae and loss of the typical dense mitochondrial granules, as well as an epigenetic modification on mitochondrial DNA [65, 66]
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Metabolomics data show that after 6 h culture, intracellular methionine level of metE mutant is only 3% of that in Wild-type (WT) strain (Zhang et al., 2023), indicating the exhaustion of methionine
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