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(PubMed) Maglione E, Marrese C, Migliaro E, et al
Together, these results indicate that decreased GSH/GSSG redox/antioxidant capacity and increased oxidative stress in the autism brain may have functional consequence in terms of a chronic inflammatory response, increased mitochondrial superoxide production, and oxidative protein and DNA damage
The form for the fourth term was chosen because the data in [27] indicate that at normal cysteine concentrations (approximately 200 M) most of the flux away from cysteine is toward GSH and only a moderate amount towards other pathways
Cardullo, R
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