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doi: 10.1038/nature12034 136 LiYYunKMuR

To explore the specific mechanism by which C5aR1 knockdown induces GBM cell death, several inhibitors were used to treat GBM cells to clarify the role of C5aR1 in regulating cell death [34]

Targeting MDSCs has therefore emerged as a promising strategy to enhance immunotherapy efficacy, and nanomedicine platforms designed to deplete or reprogram these cells are under active investigation (31, 32)

Many researchers have focused on understanding the mode of action of inhibitors that can act differently depending on the DGF-in or DFG-out conformation