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doi: 10.1007/s12032-024-02454-x 82
Nanomedicine platforms can be designed to selectively target MDSCs based on their surface receptor expression or metabolic vulnerabilities, thereby reducing systemic immunosuppression while preserving beneficial myeloid populations (32, 33)
these enzymes use endogenous adenosine triphosphate (ATP) to generate biotinyl-5-adenosine monophosphate, a mixed anhydride that reacts with lysine side chains
doi:10.1007/s12325-010-0055-0
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