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[9] Secondary hyperammonemia is caused by inborn errors of intermediary metabolism, which are characterised by reduced activity of enzymes that are not part of the urea cycle or dysfunction of cells that make major contributions to metabolism
The finding that tirzepatide binds 3-adrenoceptors with high in silico affinity modulating cAMP/cGMP balance and GLUT4 translocation in cardiomyocytes independently of canonical incretin receptor engagement (Aydos et al., 2025) illustrates that the full mechanism of action of an already-approved drug remains incompletely characterized
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