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It is therefore suggesting that the slight change of relaxation observed in the aorta without endothelium ex vivo might be due to some mechanism unrelated to the direct nitric oxide stimulation which needs further investigation
Similar concerns apply to phosphodiesterase-5 inhibitors (medications like sildenafil) and L-arginine supplements that also influence nitric oxide signaling
Sikiric et al., 2020b), in both the clinical setting (Veljaca et al., 2003
The community-derived planning ranges should not be read as clinically validated doses
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