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Based on these findings, we propose an intrinsic agonist model of the ECD for GLP-1R activation, in which the ECD is the major determining factor in activation of GLP-1R (Figure 9)
These peptides were able to pass through a lab-grown model of the intestinal barrier (Caco-2 cells)
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These side effects typically resolved on their own and were most common during or just after dose increases
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